Updated
Updated · ScienceAlert · Aug 10
Canadian Study Finds 18% Fewer Dopamine Nerve Endings in Long COVID Patients
Updated
Updated · ScienceAlert · Aug 10

Canadian Study Finds 18% Fewer Dopamine Nerve Endings in Long COVID Patients

1 articles · Updated · ScienceAlert · Aug 10

Summary

  • PET scans of 24 adults with long COVID found about 18% fewer dopamine nerve endings than in 24 healthy peers, offering visible evidence of brain changes tied to new-onset apathy and depression.
  • The losses appeared in the striatum, where reduced ventral-striatum signaling tracked lower motivation, while damage in the putamen and caudate was linked to slower movement, longer task times, memory decline and word-recall problems.
  • Researchers detected the damage in patients living with long COVID for up to 4.6 years, using a radiotracer targeting VMAT2—a biomarker already used in neurodegenerative diseases such as Parkinson's.
  • The findings may help create a diagnostic test and guide treatment: University of Toronto neurochemist Jeffrey Meyer said a dopamine-targeting drug will be tested in an upcoming clinical trial.
  • No validated tests or evidence-based treatments for long COVID currently exist, even though the condition is estimated to affect at least 65 million people worldwide.

Insights

If a respiratory infection can erase dopamine pathways, what other hidden neurological changes might long COVID leave behind?
Could the exhausting brain fog of long COVID actually be physical nerve damage deep within the brain's reward center?